Integrated Health Center
of Inland Empire
of Inland Empire
Alzheimer’s is currently affecting over 5 million Americans and is expected to double in just over 3 decades. While this disease is costing Americans over $440 billion per year, targeted pharmaceutical therapies have done nothing to delay this devastating disease.
Drug companies are trying to treat symptoms instead of the cause of the disease.
Unfortunately, the cause of Alzheimer’s has been a mystery for decades, but now, there may be hope. The most exciting recent research points to infections caused by pathogenic microbes as the trigger of Alzheimer’s and cognitive decline. These tiny bugs are linking chronic disease and cognitive decline for the first time.
Researchers diluted fluid from the brains of Alzheimer’s patients and infused that fluid into mice. This infusion “infected” mice were resulting in amyloid plaques which are the signature of Alzheimer’s disease. Now researchers are focused on these critical connections between early signs of chronic illness and later Alzheimer’s progression.
Both type 2 diabetes and AD present with local inflammation and misfolded proteins called beta-amyloidosis. These proteins develop in the presence of common microbes including Chlamydophila pneumoniae, Helicobacter pylori, and spirochetes.
Periodontitis increases the risk of Alzheimer’s, and researchers think it’s because bugs within dental plaque biofilm cross the blood-brain barrier via the bloodstream or peripheral nerves causing central nervous system inflammation linked to cognitive decline (21, 22, 23).
Herpes simplex virus type 1 (HSV1) is an widespread virus. Its estimated that around 70% of adults over 50 have had it. Almost 4 decades ago, HSV1 was suspected as a culprit in the Alzheimer’s epidemic. Over 25 years later, studies found a significant correlation between active and reactivated HSV1 and Alzheimer’s occurrence.
Atherosclerosis and Alzheimer’s share some standard features like inflammation and they’re both associated with viral and bacterial infections, including HSV1 and Chlamydophila. Besides, they both share the characteristic of plaque formation. Researchers think chronic infection causes plaque formation to protect tissues from the infection.
A strong connection has been made between early-onset Alzheimer’s and biotoxin exposure. This specific type of Alzheimer’s usually includes a combination of stroke-like speech, inability to read or interpret visual information, decreased motor skills and symptoms of Gerstmann’s syndrome. This type of Alzheimer’s is often referred to as Inhalation Alzheimer’s Disease and results alongside chronic inflammatory response syndrome (CIRS) from biotoxin exposure.
If you’re concerned about cognitive decline, it’s time to get proactive.
Identifying latent infections and supporting the immune system’s ability to fight infection and repair damage is crucial to preventing neurological damage and Alzheimer’s. Creating an effective plan starts with comprehensive diagnostics that assess your individual risk. From there, we can fix what we find and help your body function at its best.